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A current discovery highlighted a novel position for intracellular MIF in regulating the accumulation of misfolded Cu/Zn superoxide dismutase (SOD1). Mutations in SOD1 are associated with 20% of the situations of familial amyotrophic lateral sclerosis, which can be characterised by lack of motor neurons [72]. I'm sorry to say I haven't done my pla
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Even though the physiological function of MIF tautomerase enzyme exercise continues to be elusive, some tiny-molecule inhibitors targeting this site have been uncovered to become effective in interfering with MIF-receptor interactions and thus inhibited MIF-induced biological signaling. ISO-one is Among the most made use of and analyzed little mole
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